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While dogs are usually friendly and cute, they can also be dangerous at times. More often than not, dogs become dangerous due to irresponsible or abusive owners. That’s why victims of dog bites can legally pursue compensation. If you have been bitten by a dangerous, unruly dog, then you need to call or pay a visit to a dog bite attorney. California has its share of dog biting incidents everyday, and many victims are children.

Ask any dog bite lawyer in Los Angeles, and he or she will tell you how many wounds, broken bones, and even deaths they deal with every year that occur from dog biting incidents. Injuries can cost a lot of money, sometimes even several thousand dollars. Not only that, but many victims are unable to work and no longer receive an income. They need to rely on dog bite lawyers in California to help them file suit.

Even if your wound isn’t all that bad, you should still consider seeking out dog bite lawyers. Los Angeles is home to many children, and families need to be warned about dangerous dogs. The best way to do this is to ask a Los Angeles dog bite lawyer to help you get the word out.

The first cementless metal cup design implant used in hip replacement by Rush University Medical Centre (RUMC) remains extremely durable even 20 years after repeat or “revision” hip replacement.

The Harris-Galante-1 acetabular metal shell, which is designed to allow a patient’s bone to grow into the implant, remained fixed in place in 95 percent of hip revision cases at a minimum follow-up of 20 years.

The implant and its bone in-growth surface were originally developed with Jorge Galante, RUMC orthopaedic surgeon and one of the investigators. The cup’s porous surface allows bone and tissue to grow into the device to keep the hip implant in place.

Earlier generation implants relied on the use of bone cement to secure the implant to the patient’s pelvis and were associated with a higher rate of failure, particularly when used in patients who had previously had a hip implant that had failed, said an RUMC release.

“The study’s results indicate that even the first generation of this device has excellent clinical results and durability,” said Craig Della Valle, orthopaedic surgeon at Rush and study investigator. “Even after 20 years, there is low rate of failure in terms of fixation.”

Researchers previously reported the results of the Harris-Galante-1 shell for total hip revision procedures in 138 hips at a minimum of three, seven, and 15 years postoperatively. The current report presents the long-term outcomes of this group at a follow-up of 20 years.

These results were published in the February issue of the Journal of Bone and Joint Surgery.

Smoking marijuana over an extended period of time appears to greatly boost a young man’s risk for developing a particularly aggressive form of testicular cancer, a new study reveals.

In fact, researchers found that men who smoked marijuana once a week or began to use the substance on a long-term basis while adolescents incurred double the risk for developing the fastest-spreading version of testicular cancer — nonseminoma, which accounts for about 40 percent of all cases.

“Since we know that the incidence of testicular cancer has been rising in our country and in Europe over the last 40 years and that marijuana use has also risen over the same time, it seemed logical that there might be an association between the two,” said study co-author Janet Daling, an epidemiologist and member of the Fred Hutchinson Cancer Research Center’s public health sciences division in Seattle. “And when I analyzed the data, we found a fairly strong relationship with this aggressive type of testicular cancer.”

No link was found between the drug and a less aggressive and more prevalent form of the disease, known as seminoma, which strikes 60 percent of testicular cancer patients.

The findings were published in the Feb. 9 online issue of Cancer.

According to the U.S. National Cancer Institute, testicular cancer is very rare, accounting for just 1 percent of cancers among American men. Nevertheless, the disease is the most common type of cancer for American men between the ages of 15 and 34, the study noted.

Across North America, Europe, Australia and New Zealand, testicular cancer rates have increased by 3 percent to 6 percent in the past half-century. That has led some researchers to suggest that the upward trend might be the product of increased exposure among young men to one or more external factors, including a simultaneous and comparable rise in the use of marijuana.

Along those lines, the researchers noted that the testes could be particularly vulnerable to the effects of marijuana, given that the organ — along with the brain, heart, uterus and spleen — carries specific receptors for tetrahydrocannabinol (THC), the principal psychoactive ingredient in marijuana.

As well, previous human and animal research has indicated that marijuana use might lead to reduced hormonal production (particularly testosterone), poorer semen quality and impotency in men.

Daling and her team explored the notion of a marijuana-testicular cancer connection by analyzing data on 369 testicular cancer patients that had been collected by the Adult Testicular Cancer Lifestyle and Blood Specimen Study.

Participants were between the ages of 18 and 44, most were white or Hispanic, and all were residents of the Seattle-Puget Sound region. All had been diagnosed with the disease between 1999 and 2006. The men reported any history of marijuana use, as well as alcohol and smoking habits, and the same information was collected from about 1,000 healthy men.

The researchers found that current marijuana use was linked to a 70 percent increased risk for the disease.

Independent of known risk factors, nonseminoma risk was particularly high among men who used the drug at least once a week and among those who had started using it before age 18.

Though Daling emphasized that the findings are preliminary, she suggested that attention should be paid.

“We know very little about the long-term health consequences of marijuana smoking,” she cautioned. “So, although this is the first time this association has been studied and found — and the finding does need to be replicated before we are really sure what’s going on — this does give some evidence that testicular cancer may be one result from the frequent use of marijuana. And that is something that young people should keep in mind.”

But the prospect of a causal relationship between marijuana use and testicular cancer raised a lot of unanswered questions for Gary Schwartz, an associate professor in both the department of cancer biology and the department of epidemiology and prevention at Wake Forest University in Winston-Salem, N.C.

“The consensus is that most testicular cancer is thought to originate with lesions in utero, and that the peak age for testicular cancer to actually occur begins, really, right after adolescence,” he noted. “That’s when hormones released during puberty appear to promote [full-blown] cancer by essentially throwing fuel on the lesion fire, following a relatively long latency. The point being that you don’t suddenly wake up one morning with a tumor. So it’s a little hard to understand how exposure to marijuana beginning at that point could somehow play an immediate causal role.”

“But certainly, the idea that cannabis may cause cancer cells to proliferate is interesting,” Schwartz acknowledged. “It could, however, also be that recreational drug use is simply a marker for affluence, since we know that testicular cancer is traditionally a disease that is more common among the affluent. Or it could be a marker for some other event that comes along with it, that triggers lesions that lead to tumors. So, at this point, it’s just not clear to me how exactly the association between marijuana and testicular cancer would work.”